Journal CME 34.08: Changing Views of the Pathophysiology of Parkinsonism

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Journal CME 34.08: Changing Views of the Pathophysiology of Parkinsonism

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Studies of the pathophysiology of parkinsonism (specifically akinesia and bradykinesia) have along history and primarily model the consequences of dopamine loss in the basal ganglia on the function of the basal ganglia/thalamocortical circuit(s). Changes of firing rates of individual nodes within these circuits were originally considered central to parkinsonism. However, this view has now given way to the belief that changes in firing patterns within the basal ganglia and related nucleiare more important, including the emergence of burst dis-charges, greater synchrony of firing between neighboring neurons, oscillatory activity patterns, and the excessive coupling of oscillatory activities at different frequencies.

This module will take you approximately 1 hour to complete.

Faculty: Thomas Wichmann

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